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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penile enlargment pills buy place vig rx best penis enargement vig rx ingredient penis elargement patch enlargement free penile pills sample natural penis elargement hgh magna rx do penis enlarement pills really work
There are three stages of pregnancy. These are the first, second and third trimesters. The first trimester runs from week one to week fourteen, the second covers weeks 15 – 26, then the third is weeks 27 – 40. Week 1+2: This is actually before you get pregnant. It’s the stage where your body prepares itself by ovulating. And it’s in these 14 days that the egg is fertilized by the sperm Week 3: The fertilized egg now moves down the fallopian tubes, fluid passes into the ball of cells, dividing them into two. The inner cells will form your baby and the outer cells will form the placenta. Your body, at this stage, is still unaware that it is pregnant. The implantation begins as the cell ball reaches the wall of the uterus. In this process the cells actually bury into the uterus wall, which can sometimes lead to you having spotting. The implanted cell ball now becomes an embryo. Week 4: This is a week of rapid development, and your body now realises it is pregnant. The amniotic sac and cavity begin to develop and also the Yoke sac appears (this will later form the baby’s digestive system). The placenta now starts to form where implantation took place and blood from you will now go into the placenta. It is usually about day 27 that we start to feel the morning sickness. Week 5: The primitive streak (the fore runner of the brain and spinal cord) is now developing. Through this primitive streak the cells will develop into three layers: The endoderm: the bottom layer – develops the glands, lung linings, tongue, bladder, digestive tract, tonsils, urethra and associated glands. The mesoderm: the middle layer – forms the muscles, bones, heart, lungs, spleen, blood cells, and the reproductive and excretory systems. The ectoderm: the top layer – forming the skin, nails, hair, eye lens, nose, mouth, anus, tooth enamel, pituitary gland, mammary glands, and all parts of the nervous system. Other cells will be starting to develop the spine (called the notochord). The first steps towards forming the embryos head, and the first formation of the babies blood cells happen this week. Week 6: The first few days of this week is when your baby’s heart starts beating. The aorta (the largest artery in the whole body) will be starting to form at around day 40. By mid week many organs are starting to form: eyes, arm buds, liver, gall bladder, stomach and intestines, lungs and pancreas. Week 7: This is a busy week for your growing baby. During this week your baby will double in size. The lenses of the eyes are developing and there is also a recognisable tongue. The legs and arms are developing into paddles, the jaws are now visible. Week 8: The cerebellum starts to form this week. That’s the part of the brain responsible for the movement of muscles. Also hand and foot plates, elbow and wrist areas are forming. Towards the end of the eight week the hand plate has formed ridges where the fingers will be. There is further development of the eye; pigment is now appearing on the retina. Teeth buds are now forming within the gums, along with the wind pipe, bronchi, and voice box. The heart is now starting to develop the four chambers. Week 9: Your baby is now starting to form cartilage and bones. During this week the ovaries will develop into the sex organ determining whether you’re having a boy or a girl. The fingers and thumbs are now taking shape. Also the baby is now becoming more active. Week 10: It’s now that your embryo has become a baby, all be it on a rather small scale. There is a fully formed upper lip. The development of the heart now slows as it is past the critical stage. By mid week the earlobes are fully formed. Toes start to develop on the foot plate. As the bones of the palate (roof of the mouth) start to fuse together, the tongue starts to develop taste buds. Week 11: as the morning sickness starts to subside, you may feel your appetite increase. Your baby’s body starts to straighten. In males the penis is now distinguishable and in females the vagina is beginning to develop. This stage is where the baby starts to show individuality, as the muscle structure varies in each baby. Week 12: Your baby will start to develop fingernails over the next three weeks. The brain is now the same structure as it will be at birth. By the end of the week, the gall bladder and pancreas will be fully developed. Also the baby will now be opening and closing its mouth. Week 13: This week vocal chords will form in the larynx. Also the intestines will move from the umbilical cord into the abdomen, and will start to form folds and become lined with villi. Week 14: You may have noticed some changes to the areola (the area around your nipple); it may be getting larger and darker. Your baby’s heart beat will now be able to be heard using a Doppler. Breathing, sucking and swallowing motions will be being practised. The breathing practises will take the amniotic fluid in and out of the lungs. Baby’s hand also becomes more functional. Week 15: The baby’s neck is now defined, with the head now resting on the neck rather than the shoulders. The hair pattern of the baby will be defined by the 102nd day of the pregnancy your baby will now be able to turn its head, open its mouth, kick, press its lips together and turn its feet. Week 16: This week the baby’s toe nails will start to grow. The muscles will be growing stronger and the neck and head are growing straighter. As the uterus starts moving upwards you may start showing more, but this does mean less pressure on your bladder, making you feel like urinating less. Week 17: Your baby will be working on more reflexes this week; blinking, sucking, and swallowing. Development is carrying on with all the existing structures. Through the course of this month your baby’s weight will increase 6 times. Week 18: By mid week your baby’s eyes and ears will now be in the right places. The finger tips and toes will develop pads, and toe and finger prints will start to develop later in the week. Myelinization, a process of coating the nerves with a fatty substance called myelin which speeds up nerve cell transmission and insulates nerves, will start happening this week. Also by the second day of this week meconium (faecal waste) will start developing in the baby’s bowels. Week 19: A creamy looking substance that covers the baby’s body, vernix coseosa, will start to form. This protects the baby and its developing glands and sensory cells. If you’re having a baby girl primitive egg cells are now developed in the ovaries, in fact females are born with all the eggs their ovaries will ever have. Week 20: Most of the major development has now taken place, and the danger zone of the first three months is now over. Your baby will be waking and sleeping, just as newborns do. Also the formation of fine scalp hair and eyebrows will begin. Week 21: Your body is replacing the amniotic fluid very three hours at this stage of your pregnancy. Baby’s leg and arm movements increase as the muscles and bones become stronger. By the end of the week a stethoscope will be able to detect the baby’s heart beat. Week 22: If the baby is a boy, the testes will start to move from the pelvic area into the scrotum. The hair on the head and eyebrows is now visible as white and short. Week 23: The bones in the middle ear start hardening making the conduction of sound possible. The baby will start to gain some considerable weight between now and next month. The size of the baby’s body will start to get into proportion though the head will remain larger than the rest of the body. Week 24: The skin of your baby is wrinkled, but will smooth out as fat is deposited. Also by the end of this week the baby’s heart beat is so strong it is some times possible to hear it by placing an ear on your stomach. Week 25: Baby’s skin is now turning a reddish/pink as capillaries start to develop. The nostrils will now start to open, as they have been plugged unto now. The lungs will start developing blood vessels and the finger and toe nails will now be covering half the nail bed. Week 26: with the nostrils now open, muscular breathing will start. By the end of the week the lungs will be secreting surfactant, a substance which prevents the lung tissue sticking together. Also with the formation of blood vessels in the lungs, they will now also be developing air sacks. Brain wave activity starts this week for auditory and visual activity. Week 27: Bumping and thumping is becoming stronger as your baby grows stronger, you should be feeling around 10 kicks in a two hour period. Baby’s lungs are growing rapidly and there is continual development with brain patterns. Week 28: This is when the eyelids un-fuse and open up. Muscle tone is improving, and the lungs are capable of breathing air. The chances of a baby being born premature from now on, has a greatly improved chance of surviving. Week 29: Eye lashes have now grown, and although still unable to focus, baby’s eyes are now sensitive to dark and light. At this stage of pregnancy the senses of sound, smell and taste are developing. By the end of the week your baby will be able to move its eyes in their sockets. Week 30: Baby is now storing up nutrients taken in by you. Calcium for skeletal development, protein for growth and iron for blood cells. By the end of the week the languno (the small hairs that covered the baby’s body), is nearly all gone apart from some patches on the shoulders and back. Week 31: As the actual growth starts to slow down, the internal organs are still maturing, so make sure your still getting enough folic acid, iron and calcium. Should your baby be born this week they would have the ability to breath, see, listen learn and remember. Week 32: The baby’s iris is now reacting to light. All five senses are now registering with your baby, although smell is limited as baby can’t breathe air in the uterus. Week 33: your baby may now be sucking its fingers. Constipation could be starting for you as your uterus puts more and more pressure on your bowels. Week 34: The pigment of the eyes is not quite fully developed yet, this leaves the eyes looking blue regardless of final colour. And this week your baby will start to develop its own immune system. Week 35: In baby boys the decent of the testes will complete any time now. Your baby may now shift into your pelvis in a head down position, but not all babies’ do this before birth. Week 36: Dimples on the elbows and knees will be forming as well as creases in the neck area due to continual deposits of fat. Also this fat will help baby maintain its body temperature. Week 37: Around 85% are born within two weeks of their actual due date (either before or after), so as you enter this stage be aware for signs of labour. The baby is practising being more aware of its surroundings; this is the ‘orientating response’. This is where the baby will turn towards any source of light. The end of this week marks the end of development, growth will now slow down. Week 38: Meconium is accumulating in the intestines. Meconium is a dark green mass of waste product and cells from the gall bladder, liver and pancreas. Although shortly after birth this will all come out. Week 39: as the baby is settling into your pelvis, you maybe feeling clumsy and off balance. This is because your centre of gravity shifts. Make sure you’re prepared for your trip to the hospital. Week 40: welcome to the final week, that’s if you have not given birth already. Your body will be giving the baby antibodies so it can protect its self from many diseases. The baby will finish dropping into its resting place before birth. So congratulations and welcome to your new born child. pnis enlargement stretcher penis elargement system vimax natural penis enlargement penis enlargment program penis enlargment vimax penis enlargement pills product truth about penile enlargment permanent penis enlargment do penis enlarement pills really work
Penis curvature is one of those unpleasant problems plaguing men, filling them with anxiety and driving away peace of mind. A visible curve in the penis is bound to make any man less eager to take down his pants in front of a lady. What if she starts laughing? What if she recoils from it? Nobody likes to be seen as a freak or as something to be laughed about. Although the man in question is in no way responsible for this problem, others can’t help but see this as a failure or as a dubious physical feature. On the other hand, some men are actually proud of their special look. A curved penis serves to underscore a rebellious or outré personality that refuses to bow to tradition. Moreover, a bent penis can be very desirable for women, too, because the curve allows the head of the penis to rub against areas not normally touched by a straight penis. Thus, it may be said that a special penis is not seen as a liability in certain circles. However, one should weigh carefully the pros and cons of a curved penis because most women still prefer a straight, normal-looking penis over a bent one. Curvatures are caused either by the natural growth of the penis or by a condition knows as Peyronie’s disease. The natural curvature occurs when the two chambers of the penis develop at different rates through the years, thus bending the penis toward the slower developing chamber. There is no way that such a natural development tendency can be forecast and prevented, although it can be corrected whenever the user wants it. However, most men whose penises are bent refuse to seek the treatment that is within easy reach and would rather take their chances with the kindness of women. Peyronie’s disease is a condition named after Francois de la Peyronie, surgeon to Louis XIV of France, who was the first man to describe a treatment in 1743. While the cause of this disease is not well understood, its mechanism is perfectly clear. The “tunica albuginea” is the tough layer of connective tissue that contains the sponge-like Corpora Cavernosa. For reasons unknown, scar tissue begins to form in the “tunica albuginea”, which prevents the normal expansion of the penis during erections and, in time, bends the penis to one side or the other. The fully natural way of straightening the penis is to use penis exercises. Penis fitness programs offer exercises that can break the scar tissue down and help reverse the curve. The Jelq is one such penis exercise that has been successfully used to address this problem. Another good way of dealing with a curved penis is to use a traction device. The constant pull of the traction device is bound to loosen up the “tunica albuginea” and straighten the penis. Actually, your best bet is to try both of these two penis straightening techniques at the same time. This will speed up the healing process, although you have to realize that no miracle is going to happen over night. The scar tissue took a long time to form and is not going away fast. What you need is patience and persistence, the two tools that will take you to your goal. Exercise and wear the traction device everyday and you will see the bend disappearing little by little.